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目的通过对导致未足月胎膜早破的生殖道病原微生物的分析,从而指导临床抗生素应用。方法试验组为106例,采用血培养基、泌尿生殖道病原性支原体药敏定量培养板对PPROM孕妇宫颈分泌物进行一般细菌和解脲支原体(UU)检测,同时分别选择106例足月正常孕妇作为对照。结果 PPROM孕妇宫颈分泌物细菌阳性率(22.64%)明显高于正常孕妇(4.72%)(P<0.05),依次为耐氨苄西林大肠埃希菌、产气肠杆菌、耐甲氧西林金黄色葡萄球菌、粪肠球菌、表皮葡萄球菌、铜绿假单胞菌、念珠菌等;PPROM病例解脲支原体(UU)阳性率高达39.62%,明显高于正常孕妇(16.98%),两者之间差异有统计学意义(P<0.01)。结论在未足月胎膜早破(PPROM)患者中耐氨苄西林大肠埃希菌、产气肠杆菌、耐甲氧西林金黄色葡萄球菌、粪肠球菌、表皮葡萄球菌、铜绿假单胞菌、念珠菌检出率增多;解脲支原体仍是未足月胎膜早破的重要致病因素。
Objective To guide the clinical application of antibiotics by analyzing the genital pathogenic microorganisms that cause premature rupture of membranes. Methods The experimental group consisted of 106 cases. The blood culture medium and pathogenic mycoplasma mycoplasma were used to detect the general bacterial and UU (Mycoplasma urealyticum) secretions in pregnant women with PPROM. At the same time, 106 full-term pregnant women were selected as normal pregnant women Control. Results The positive rate of cervix secretion in PPROM pregnant women (22.64%) was significantly higher than that in normal pregnant women (4.72%) (P <0.05), followed by E. coli strains resistant to ampicillin, Enterobacter aerogenes and methicillin- Enterococcus faecalis, Staphylococcus epidermidis, Pseudomonas aeruginosa, Candida albicans, etc. The positive rate of Ureaplasma urealyticum (UU) in PPROM cases was 39.62%, which was significantly higher than that of normal pregnant women (16.98%), the difference was Statistical significance (P <0.01). Conclusions In patients with incomplete premature rupture of membranes (PPROM), the resistance to ampicillin-resistant Escherichia coli, Enterobacter aerogenes, methicillin-resistant Staphylococcus aureus, Enterococcus faecalis, Staphylococcus epidermidis, Pseudomonas aeruginosa, Candida albicans detection rate increased; Ureaplasma urealyticum is still not enough on the premature rupture of membranes an important risk factor.