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目的 观察吸入液氧对高原人体力竭运动血液流变学的影响。 方法 对进驻海拔 370 0m高原3个月的 10名健康青年在吸入液氧 (吸氧组 ,4L/min)和不吸液氧 (对照组 )的条件下 ,采用功率自行车进行递增负荷运动 ,并在安静时和运动后测定血中天冬氨酸氨基转移酶 (AST)、乳酸脱氢酶 (LDH)、α 羟丁酸脱氢酶(α HBDH)、肌酸激酶 (CK)及其同功酶 (CK MB)活性。 结果 与安静时比较 ,对照组和吸氧组运动后AST、LDH、α HBDH、CK及CK MB均增高显著 (P <0 0 5或P <0 0 1)。运动后吸氧组较对照组AST、LDH、α HBDH、CK及CK MB降低显著 (P <0 0 5或P <0 0 1)。 结论 吸入液氧能延长供氧时间 ,对心肌细胞损伤有保护作用 ,使细胞膜通透性降低 ,减少心肌酶释放
Objective To observe the effects of inhaled oxygen on hemorheology of exhaustive exercise in high altitude. Methods Ten healthy youths who were stationed at an elevation of 370 0 m for 3 months were subjected to incremental load exercise with power bikes under inhalation of liquid oxygen (oxygen inhalation group, 4 L / min) and non-absorbing liquid oxygen (control group) Blood levels of aspartate aminotransferase (AST), lactate dehydrogenase (LDH), α-hydroxybutyrate dehydrogenase (α HBDH), creatine kinase (CK) and their homokines were measured at rest and after exercise Enzyme (CK MB) activity. Results Compared with resting, the levels of AST, LDH, α HBDH, CK and CK MB in control group and oxygen inhalation group increased significantly (P <0.05 or P <0.01). After exercise, the levels of AST, LDH, α HBDH, CK and CK MB in the oxygen inhalation group decreased significantly (P <0.05 or P <0.01). Conclusion Oxygen inhalation can prolong the oxygen supply time and protect the injured cardiomyocytes, decrease the cell membrane permeability and decrease the release of myocardial enzymes