吸入盐酸可以损伤肺泡-毛细血管膜,导致肺血管阻力和通透性增加,引起急性肺水肿。β肾上腺能兴奋剂不仅可以降低肺血管阻力,还可降低血管通透性。本文作者给兔气管内滴入0.1N 盐酸2ml/kg,造成酸损伤型肺水肿模型,研究异丙去甲肾上腺素的作用机制。将动物分为5组:①无酸对照组(4只):不滴 Hydrochloric acid inhalation can damage the alveolar - capillary membrane, leading to increased pulmonary vascular resistance and permeability, causing acute pulmonary edema. Beta adrenergic stimulants not only reduce pulmonary vascular resistance, but also reduce vascular permeability. The author of rabbit into the tracheal instillation of 0.1N hydrochloric acid 2ml / kg, resulting in acid injury model of pulmonary edema, the mechanism of action of isoproterenol. The animals were divided into 5 groups: ① acid-free control group (4): do not drip