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据1955年1956年Rachmilewitz氏及他氏等的报道在急性肝臟疾患中曾发現血淸維生素乙12数値升高,偶然在肝硬化者亦然。急性肝炎的急性期当肝細胞崩解非常明显时,血清維生素乙12数値升高。因此假定,此系受損害的肝細胞將儲留在肝內的維生素乙12釋放出至血流中。这假定在被用四氯化碳而促使狗发生急性肝臟坏死的中所証实。据1956年Stein氏及他氏的实驗,在这些狗中当四化碳以門靜脈內投藥后1—2小时,卽发現
According to 1955 1956 Rachmilewitz’s and his reports, etc. in acute liver disease has found that blood urea nitrogen, vitamin B12 increased, occasionally in cirrhotics. The acute phase of acute hepatitis when the disintegration of liver cells is very obvious, the serum vitamin B12 increased. Therefore, it is assumed that the damaged liver cells release vitamin B12, which is stored in the liver, into the bloodstream. This is presumed to be evidenced by the use of carbon tetrachloride to induce acute liver necrosis in dogs. According to Stein’s and his experiments in 1956, in these dogs 1-2 hours after tetralin was given intravenously in the portal vein,