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已知甲状腺功能异常导致血液胆固醇(Ch)浓度有变化。体外试验或临床研究都酶说明脂质代谢的调节受甲状腺激素的影响。的活化可调节脂蛋白的合成和分解,加速组织中Ch的转化与消除,改变了动脉粥样硬化的进程。密切依赖甲状腺的特异性低密度脂蛋白(LDL)受体的活性为甲状腺激素的周围生物活性提供了一个敏感的定量指标。
Thyroid dysfunction is known to cause changes in blood cholesterol (Ch) concentrations. In vitro or clinical studies indicate that the regulation of lipid metabolism is affected by thyroid hormones. Activation can regulate the synthesis and decomposition of lipoprotein, accelerate the transformation and elimination of Ch in tissues and change the process of atherosclerosis. Closely dependent thyroid specific LDL receptor activity provides a sensitive quantitative indicator of the peripheral biological activity of thyroid hormones.