论文部分内容阅读
观察了耐力游泳训练对大鼠骨骼肌和肝脏金属硫蛋白（Ｍｅｔａｌｌｏｔｈｉｏｎｅｉｎ，ＭＴ）诱导性的影响，以及训练大鼠力竭性游泳后，ＭＴ和锌、铜、铁、锰、钙、镁等金属离子含量的动态变化，探讨了它们之间的关系。结果显示：耐力性游泳训练可使大鼠骨骼肌和肝脏ＷＴ的基础水平升高。训练大鼠力竭性游泳后，骨骼肌和肝脏中诱导合成的ＷＴ的峰值提前出现，表明运动训练可加快ＭＴ的诱导合成。ＷＴ较高的基础水平和快速的诱导合成，可能是训练促进运动恢复的生理机制之一。训练大鼠力竭性游泳后，骨骼肌锌、铜、锰和肝脏锌、铜、锰、铁的变化趋势与ＷＴ的变化趋势较为一致，表明ＷＴ可能参与了这些金属离子的代谢。ＷＴ可能通过调节金属离子代谢，在机体的运动恢复中发挥重要作用。
The effects of endurance swimming training on the induction of MT and MT in rat skeletal muscle and liver were observed, and MT and zinc, copper, iron, manganese, calcium, magnesium and other metals Ion content of the dynamic changes, to explore the relationship between them. The results showed that endurance swimming training increased basal levels of WT in skeletal muscle and liver of rats. After training exhaustive swimming in rats, the peak of induced WT in skeletal muscle and liver appeared earlier, indicating that exercise training can accelerate the induction of MT synthesis. Higher basal level of WT and rapid induction of synthesis may be one of the physiological mechanisms by which training promotes motor recovery. After exhaustive swimming training, the changes of zinc, copper, manganese and iron in zinc, copper, manganese and iron of skeletal muscle were consistent with those of WT, indicating that WT might be involved in the metabolism of these metal ions. WT may play an important role in the body’s motor recovery by regulating the metabolism of metal ions.