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我院10年来收治肾小管酸中毒(RTA)32例,诊断依据为:①酸中毒;②低血钾、低血钙、低血磷及(或)高尿钙、高尿磷,血清碱性磷酸酶(AKP)升高,继发骨及肾脏病变(骨疏松、软化,肾钙化、结石等);③肾小管功能异常证据。临床资科女25例,男7例,年龄17～64岁。病程最长13年。13例有原发病,以肾盂肾炎最多,19例无原发病发现。临床表现以烦渴多饮、食欲减退、乏力、肌病、心律失常、骨关节痛多见,其它包括手足搐搦、癫痫、生长发育障碍、泌尿系结石、牙齿脱
In our hospital 10 cases of renal tubular acidosis (RTA) in 32 cases, the diagnosis is based on: ① acidosis; ② hypokalemia, hypocalcemia, hypophosphatemia and (or) high urinary calcium, high urinary phosphorus, serum alkaline Phosphatase (AKP) increased secondary bone and kidney disease (osteoporosis, softening, renal calcification, stones, etc.); ③ evidence of tubular dysfunction. Female 25 cases of clinical data, male 7 cases, aged 17 to 64 years. The longest course of 13 years. Thirteen patients had primary disease, with the highest incidence of pyelonephritis and 19 patients without primary disease. Clinical manifestations of thirst and polydipsia, loss of appetite, fatigue, myopathy, arrhythmia, joint pain more common, other including tetany, epilepsy, growth and development disorders, urinary stones, dental off