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心跳骤停/心肺复苏(CA/CPR)所引起的能量代谢障碍主要与线粒体的结构与功能受损有关,线粒体功能障碍是中枢神经系统损伤的重要机制[1]。脑缺血再灌注后细胞能量代谢的恢复是细胞其他功能恢复的基础,若细胞能量代谢障碍持续存在,则必然导致细胞凋亡或坏死[2-3]。本研究探讨了心?
Cardiac arrest / cardiopulmonary resuscitation (CA / CPR) caused by energy metabolism disorders are mainly related to the structure and function of mitochondria damage, mitochondrial dysfunction is an important mechanism of central nervous system injury [1]. Recovery of cellular energy metabolism after cerebral ischemia-reperfusion is the basis for the recovery of other functions of the cell. If the cell’s energy metabolism disorder persists, it will inevitably lead to cell apoptosis or necrosis [2-3]. This study explores the heart?