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以心肌缺血再灌注损伤的病理生理现象为前提,从自由基损伤、钙超载、能量代谢、细胞因子、细胞凋亡五个方面去阐述心肌缺血再灌注损伤的主要机制及药物治疗的研究进展。
Myocardial ischemia-reperfusion injury pathophysiological phenomena as the premise, from the free radical damage, calcium overload, energy metabolism, cytokines, apoptosis of myocardial ischemia and reperfusion injury to explain the main mechanism of drug treatment progress.