心脏骤停(CA)及心肺复苏(CPR)过程中,机体经历完全血液循环停止、严重缺氧、酸中毒和缺血/再灌注等一系列的应激过程,大量神经内分泌及细胞因子参与这一过程,这些因子相互作用,对CA的病理生理改变、复苏药物的应用效果及CPR的预后产生重要影响,而CA及CPR后甲状腺功能变化及其应 During cardiac arrest (CPR) and cardiopulmonary resuscitation (CPR), the body undergoes a series of stress processes such as complete blood circulation stop, severe hypoxia, acidosis and ischemia / reperfusion. A large number of neuroendocrine and cytokines participate in this process A process, the interaction of these factors, the pathophysiology of CA changes, the application of resuscitative drugs and prognosis of CPR have a significant impact, and CA and CPR after thyroid function changes and its