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患者女,20岁。于1989年4月17日自服重铬酸钾约20g,被发现后立即送当地医院洗胃,24小时后出现无尿(尿量<50ml/d),曾先后用甘露醇,大剂量速尿(>900mg/d)静脉注射,以及其它内科疗法均不能凑效,2天后转入我院。体检:血压波动于14.3～18.6/9.3～11.9kpa,神清,精神差,全身明显浮肿,频繁恶心、呕吐,且伴有上消化道出血,心慌,气促,两肺呼吸音粗糙,心率90～120次/分。血尿素氮由11mmol/L上升至17mmol/L,血肌酐由298μmol/L上升至390μmol/L。于无尿第四天行腹部插管透析,先后行间歇性腹膜透析(IPD)和持续性非卧床腹膜透析(CAPD)共26天。患者水肿逐渐消退,恶心呕吐消失,上
Female patient, 20 years old. On April 17, 1989 self-serving potassium dichromate about 20g, was found immediately after the local hospital gastric lavage, 24 hours after the emergence of anuria (urine output <50ml / d), has successively with mannitol, high-dose speed Urine (> 900mg / d) intravenous injection, and other medical treatment can not be effective, 2 days later transferred to our hospital. Physical examination: blood pressure fluctuations in the 14.3 ~ 18.6 / 9.3 ~ 11.9kpa, Shen Qing, poor spirits, systemic edema, frequent nausea and vomiting, accompanied by upper gastrointestinal bleeding, palpitation, shortness of breath, rough breathing sounds of both lungs, heart rate 90 ~ 120 beats / min. Blood urea nitrogen rose from 11mmol / L to 17mmol / L, serum creatinine increased from 298μmol / L to 390μmol / L. Dialysis was performed on the fourth day of anuria for abdominal intubation, followed by intermittent peritoneal dialysis (IPD) and continuous ambulatory peritoneal dialysis (CAPD) for 26 days. Patients edema subsided, nausea and vomiting disappeared, on