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多巴胺与多巴酚丁胺均能使慢性心衰患者心排血量增加,但两药对左心室灌注压、肺毛细血管楔压(PCWP)的影响不同,多巴胺增加心排血量时使PCWP上升,而多巴酚丁胺则使 PCWP 下降。由于PCWP 轻微增高可加剧肺水肿,为此作者观察了急性呼吸衰竭者用多巴胺与多巴酚丁胺后血流动力学的变化,以便为选用该类药物提供理论依据。8例急性呼吸衰竭作辅助通气的患者,胸片示弥漫性肺浸润,吸入氧浓度需达50%以上。用多巴胺或多巴酚丁胺静脉滴注,滴注初速为2.5μg/kg/min,逐渐加量直至达到以下指标之一时,则停止给药①血压平均升高25mmHg;②心排血量增加25%;③心率
Dopamine and dobutamine can make patients with chronic heart failure cardiac output increased, but the two drugs on left ventricular perfusion pressure, pulmonary capillary wedge pressure (PCWP) of different effects, dopamine increased cardiac output when PCWP While dobutamine reduced PCWP. Because PCWP slightly increased may aggravate pulmonary edema, for which the authors observed the acute respiratory failure with dopamine and dobutamine hemodynamic changes, in order to provide a theoretical basis for the selection of such drugs. 8 cases of acute respiratory failure for assisted ventilation in patients with chest X-ray showed diffuse pulmonary infiltration, inhaled oxygen concentration to be up to 50%. With dopamine or dobutamine intravenous drip, the initial infusion rate of 2.5μg / kg / min, and gradually increase until one of the following indicators, then stop the administration ① blood pressure increased by an average of 25mmHg; ② cardiac output increased 25%; ③ heart rate