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我们过去已报告 CCK 可能参于脑缺血病理生理过程。为进一步研究其机制,我们观察了脑缺血蒙古沙土鼠离体皮层脑片 CCK 基础释放和钾离子存在下释放的改变,同时观察神经节苷脂是否能翻转这些变化。双侧结扎蒙古沙土鼠颈动脉半小时后,迅速取出
We have reported in the past that CCK may be involved in the pathophysiological process of cerebral ischemia. To further investigate its mechanism, we observed changes in CCK release and potassium ion release in isolated cortical brain slices of Mongolian gerbils with cerebral ischemia, and observed whether gangliosides could reverse these changes. Both sides of the Mongolian gerbils carotid artery ligation half an hour later, quickly removed