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应激反应引起急性胃粘膜损害的机制认为与胃粘膜的微循环功能紊乱，胃分泌状态的变化和胃运动异常有关。胃粘膜血液循环和调节中“自主调节脱逸”（ａｕｔｏｒｅｇｕｌａｔｉｏｎｅｓｃａｐｅ）的存在，使胃粘膜的血液供应在应激反应时会出现缺血－再灌注过程，从而产生大量的氧自由基，对胃粘膜组织造成损害。
The mechanism of stress-induced acute gastric mucosal damage is believed to be related to dysfunction of the microcirculation of the gastric mucosa, changes in gastric secretion status, and gastric motility abnormalities. Gastric mucosa blood circulation and regulation of “autoregulation escape” (autoregulationescape) the existence of the gastric mucosal blood supply in response to stress occurs when the ischemia-reperfusion process, resulting in a large number of oxygen free radicals, the gastric mucosa Organization caused damage.