急性冠状动脉综合征 (ACS)是由于动脉粥样硬化斑块发生裂隙 ,并在此基础上形成血栓而使冠状动脉部分或完全闭塞所致。以往的治疗非常强调ACS的最初数天内给予强化抗凝治疗 ,并取得了很好的疗效。但有证据表明 ,ACS发生后的 4～ 6周血栓形成事件的危险仍继续增加[1] ,凝血酶和血 Acute coronary syndrome (ACS) is due to atherosclerotic plaque cracks, and on the basis of the formation of thrombus and coronary artery due to partial or complete occlusion. In the past, treatment was emphasized on the first few days of ACS to give intensive anticoagulant therapy, and achieved good results. However, there is evidence that the risk of thrombotic events continues to increase 4 to 6 weeks after ACS [1], thrombin and blood