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哈佛大学医学院科研人员曾成功利用小鼠为模型研究并揭示了多种肿瘤发病的分子机理和遗传学机制,包括白血病、淋巴瘤以及前列腺癌等实体肿瘤。最近,在进一步的研究中,研究小组又提出了一种关于信使RNAs如何转录假基因,以及长链非编码RNAs如何相互“交流”的新假说,将有助于癌症等疾病的分子机理解析。还提出了一个关于肿瘤抑制基因TSG功能的新模型,用以解释
Harvard Medical School researchers have successfully used mice as a model to study and reveal a variety of tumor pathogenesis molecular mechanisms and genetic mechanisms, including leukemia, lymphoma and prostate cancer and other solid tumors. Recently, in a further study, the team proposed a new hypothesis on how messenger RNAs transcribe pseudogenes and how long-chain, non-coding RNAs interact with each other, which will contribute to the molecular mechanisms of cancer and other diseases Analysis. A new model for the function of tumor suppressor TSG was also proposed for explanation