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本文应用日本Atoka公司产浅表超声仪对缺碘地区长期供碘后和非缺碘地区甲状腺形态进行了各个切面的描记。结果是;居住在缺碘地区人群,甲状腺异常改变率占28.67%,而非缺碘地区为9.94%。二者差异非常显著,缺碘与非缺碘地区甲状腺改变异常率均女高于男。异常改变主要发生在>30岁年龄组;而<30岁年龄组则无差异。原因是从1958年后缺碘地区开始供应碘盐,机体缺碘状态已被纠正,而>30岁年龄组是供碘盐前出生并居住在病区,是因缺碘而造成的甲状腺损害,致病后虽供应了碘盐,然而甲状腺内部却残留了不同程度的病理损害,这些损害主要是形成腺瘤样甲状腺肿、结节性甲状腺肿、甲状腺钙化、甲状腺恶变及桥本氏病等。
In this paper, Japan’s Atoka company produced superficial ultrasonic instrument in iodine-deficient areas after long-term supply of iodine and non-iodine-deficient areas of the thyroid morphology of the various sections of the description. The result was that residents living in areas of iodine deficiency accounted for 28.67% of the abnormalities in the thyroid gland, compared with 9.94% in non-iodine-deficient areas. The difference between the two was very significant. The abnormal rates of thyroid changes in both iodine-deficient and non-iodine-deficient areas were higher than those in male. Abnormal changes occurred mainly in> 30-year-old age group; <30-year-old age group no difference. The reason is that iodized salt is supplied from areas lacking iodine after 1958, and the iodine deficiency status of the body has been corrected. However, the 30-year-old group was born before the salt iodization and lived in the ward because of thyroid damage caused by iodine deficiency. Although the supply of iodized salt after the disease, however, the thyroid gland remains within the varying degrees of pathological damage, the main damage is the formation of adenomatous goiter, nodular goiter, thyroid calcification, thyroid malignant and Hashimoto’s disease.