论文部分内容阅读
本文探讨了细胞因子外源性ｒＩＬ－１和ｒＩＬ－２对机体内污染荧光涂料激发能源１４７ｐｍ时的辐射防护作用以及喹胺酸促排１４７ｐｍ的效果。研究发现，１４７ｐｍ可使中枢免疫器官骨髓和胸腺细胞的３Ｈ－ＴｄＲ掺入反应，以及外周免疫器官脾Ｂ淋巴细胞ＬＰＳ转化反应受到抑制，ＤＮＡ合成显著减少，而外源性，ｒＩＬ－１与ｒＩＬ－２则可完全或部分逆转１４７ｐｍ损伤骨髓细胞、胸腺细胞和脾Ｂ淋巴细胞的增殖抑制能力，对１４７ｐｍ损伤机体呈现明显的辐射防护作用。而喹胺酸对１４７ｐｍ则有明显的加速排除效果。
This article explored the radioprotective effects of exogenous cytokines, rIL-1 and rIL-2, on the 147 pm excitation energy of fluorescent contaminants in vivo and 147 pm stimulation of quetiapine. The results showed that 147pm could induce the incorporation of 3H-TdR into the bone marrow and thymocytes of the central immune organs and inhibit the LPS transformation of splenic B lymphocytes in peripheral immune organs and significantly reduce the DNA synthesis. However, exogenous rIL-1 and rIL -2 can completely or partially reverse the 147pm injury of bone marrow cells, thymocytes and splenic B lymphocyte proliferation inhibitory ability of 147pm injury showed significant radiological protective effect. Quinic acid on 147pm significantly accelerated the exclusion effect.