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在大量或快速胸腔抽气或抽液后可发生肺水肿,其主要的促发因素是肺组织原先受较长时期(>3d)的压缩蒌陷,和使用过大的负压吸引。发病机制可能主要由于长期肺蒌陷的缺氧,使肺毛细血管内皮损伤,而致血管通透性增加所致。警惕本病,诊断不难,但需有胸部 X 线单侧性肺水肿的证据。治疗原则同一般非心源性肺水肿,但预后较之为佳。采取一定措施,本病可以预防。
Pulmonary edema can occur after a plethora of rapid or pleural aspirates or pump-outs, with the primary contributor to the collapse of the lung tissue that was previously compressed by longer periods of time (> 3d) and by the use of excessive negative pressure. Pathogenesis may be mainly due to hypoxia caused by long-term lung depression, pulmonary capillary endothelial damage, resulting in increased vascular permeability caused. Be wary of the disease, the diagnosis is not difficult, but need to have evidence of unilateral chest X-ray edema. The same principles of treatment with non-cardiogenic pulmonary edema, but the prognosis is better. Take certain measures, the disease can be prevented.