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除少数有心肺窘迫症状但无先天心病者不易辨认外,新生儿呼吸窘迫的中央型青紫通常不准与先天性心脏病所致者相鉴别,本文介绍应用对比超声心动图,能检出经卵园孔的心房水平右向左分流,以资区别这两种产生青紫的不同病因。本组包括20例足月新生儿。其中A组共10例,出生体重正常,没有紫绀和呼吸或心血管功能障碍,但有高胆红素血症需要换血。B组10例在出生24小时内无先天性心脏病表现,但有紫绀和严重的呼吸窘迫,体重自3.0～4.1kg,在吸入100%纯氧时PO_2为20～40mmHg。所有患儿都有心力衰竭表现,如气促、心动过速、奔马律、心脏扩大和肝肿大。且至少存在一种促使肺动脉高压的因素,如低血糖、低温、红细胞增多、新生儿窒息和原发的肺部疾病。
In addition to a small number of patients with symptoms of cardiopulmonary distress but no congenital heart disease who are not easy to identify, the neonatal respiratory distress of central cyanosis are usually not allowed and congenital heart disease were identified, this article describes the application of contrast echocardiography, can be detected by the egg Park hole atrial level right to left shunt, in order to distinguish between the two different causes of bruising. The group includes 20 full-term newborns. A group of 10 cases, normal birth weight, no cyanosis and respiratory or cardiovascular dysfunction, but hyperbilirubinemia need to exchange blood. In group B, 10 patients had no congenital heart disease within 24 hours of birth, but had cyanosis and severe respiratory distress. The body weight ranged from 3.0 to 4.1 kg. PO_2 was 20 to 40 mmHg when 100% pure oxygen was inhaled. All children had heart failure manifestations such as shortness of breath, tachycardia, gallop, heart enlargement and hepatomegaly. And there is at least one factor that causes pulmonary hypertension, such as hypoglycemia, hypothermia, polycythemia, neonatal asphyxia and primary lung disease.