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犬在失血休克５ｈ内，ＰａＯ＿２波动上升，ＰａＣＯ＿２进行性降低。用ＰｖＯ＿２和ＰｖＣＯ＿２反映细胞用氧状态发现，在休克１ｓｔ至３ｒｄｈ在心房及诸器官有进行性ＰｖＯ＿２降低和ＰｖＣＯ＿２升高，在３ｔｄ至５ｔｈｈ二者相对恒定。此时从血氧解离曲线看，静脉氧饱和度和张力仍超过代偿极限１～２倍，代偿储备远未耗竭，氧的供求平衡是一种假象，实为细胞有用氧障碍所致。将诸器官作双因素方差分析发现肝ＰｖＯ＿２最低ＰｖＣＯ＿２最高，而肾则相反，变化反映了器官的解剖生理特点，肾ＰｖＣＯ＿２从３ｒｄｈ方急剧上升，提示开始有肾排酸保硷功能受损。在５ｈ实验全过程，动脉血ｐＨ略高于静脉血ｐＨ并平行地进行性下降，未随血气稳定而不变，说明它不是缺氧引起。
Dogs within 5h after hemorrhagic shock, PaO_2 fluctuations increased, PaCO_2 progressively decreased. Using PvO_2 and PvCO_2 to reflect the oxygen status of the cells, it was found that progressive PvO_2 decreased and PvCO_2 increased in the atria and organs during the first to third shock of shock, and remained relatively constant between 3td and 5thh. At this point from the blood oxygen dissociation curve, venous oxygen saturation and tension is still more than the compensation limit of 1 to 2 times, far from the depletion of compensation reserves, oxygen supply and demand balance is an illusion, in fact, due to cellular oxygen disorders . The results of two-factor analysis of variance showed that the lowest PvCO 2 in liver was the highest, while the opposite was found in kidney. The changes reflected the anatomical and physiological characteristics of organs. The renal PvCO 2 increased sharply from 3rdh, suggesting impaired renal acid secretion. In the whole process of 5h experiment, arterial blood pH slightly higher than the venous blood pH and decreased in parallel, not unchanged with the blood gas unchanged, indicating that it is not caused by hypoxia.