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患儿女,13岁,以自服异烟肼8000mg,抽风、昏迷5h 于1988年10月1日急诊入院。入院前曾在当地乡卫生院给清水洗胃,住院后再次洗胃,洗出为棕色液体。查体:脉搏细弱,68次/min,BP10.66/8.00kPa,压眶上神经无反应,呈深昏迷状,频繁抽风,呼吸急促,面色青灰,口唇及四肢末梢发绀,皮肤发花,双瞳孔忽大忽小,右>左,波动在2～6mm 之间,光反应消失。双肺闻及大量痰鸣,心、腹(-)。布氏征、克氏征及巴氏征均(-)。急查k~+为3.65mmol/L,Na~+为142mmol/L,Ca~(2+)为2.25mmol/L,Co_(2-)CP12.44mmol/L,(?)UEN5.71mmol/L;CKG 示下壁心肌受损。入院后按急性异烟肼中毒、脑水肿、脑疝形成、
Children with children, 13 years old, self-serving isoniazid 8000mg, ventilation, coma 5h in October 1, 1988 emergency admission. Before admission to the local township hospitals to clean water gastric lavage, gastric lavage again after hospitalization, washed out as a brown liquid. Examination: weak pulse, 68 times / min, BP10.66 / 8.00kPa, no pressure on the supraorbital nerve, was deep coma, frequent ventilation, shortness of breath, pale blue, lips and limbs, cyanosis, Pupil suddenly big suddenly small, right> left, fluctuating between 2 ~ 6mm, light reaction disappeared. Lung smears a lot of phlegm, heart, abdomen (-). Brinell’s sign, Krypton sign and Pakistan’s sign (-). The results showed that there was no significant difference between the control group and the control group (p> 0.05). The maximal values of k ~ + were 3.65mmol / L, Na ~ + 142mmol / L, Ca 2+ 2.25mmol / L, Co_ (2-) CP12.44mmol / L, ; CKG showed inferior myocardial damage. After admission, according to acute isoniazid poisoning, cerebral edema, cerebral hernia formation,