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为了探讨寡糖诱导物的分子结构与诱导抗病性之间的相互关系,以7个化学合成的寡糖为诱导物,研究了烟草植株对黑胫病的诱导抗性。结果表明,-β1,3-乙酰氨基葡聚四糖、-β1,4-乙酰氨基葡聚三糖和-β1,4-乙酰氨基葡聚四糖的诱导处理对烟草黑胫病表现活体抗性,相对诱导效果分别为62.5%,50.0%和75.0%;-β1,3-乙酰氨基葡聚四糖、-β1,4-乙酰氨基葡聚三糖和-β1,4-乙酰氨基葡聚四糖的诱导处理对烟草黑胫病表现离体抗性,相对诱导效果分别为56.25%,50.0%和62.5%。研究结果表明寡糖的聚合度以及寡糖主链的糖苷键连接方式可能是影响诱导抗病性的重要因子,不同浓度寡糖处理后烟草的诱导抗病性有差异。
In order to investigate the relationship between the molecular structure of the oligosaccharide inducer and the induced disease resistance, seven chemically synthesized oligosaccharides were used as inducers to study the induced resistance of tobacco plants to black shank. The results showed that induction treatment of -β1,3-acetamidotetraose, -β1,4-acetylglucosaminoglycotriose and -β1,4-acetamidoglucotetraose showed live resistance to tobacco black shank disease , The relative induction effects were 62.5%, 50.0% and 75.0%, respectively; β1,3-acetylglucosaminopolyotetraose, -β1,4-acetylglucosaminoglycotriose and -β1,4-acetylglucosaminopolyotetraose Induced resistance to tobacco black shank disease in vitro resistance, the relative induction effect were 56.25%, 50.0% and 62.5%. The results showed that the degree of polymerization of oligosaccharides and glycosidic linkage of oligosaccharides backbone may be an important factor affecting the resistance to disease resistance, different concentrations of oligosaccharides treated tobacco induced disease resistance differences.