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本文对23例新生儿窒息及呼吸衰竭患儿采用高效液相色谱-电化学检测法测定去甲肾上腺素（NE）和肾上腺素（E），10例正常脐血为对照组，结果显示，窒息及呼衰缺氧可导致交感神经系统活性增强，分泌NE与E明显增多，急性期87．51±35．31pg／ml与恢复期38．55±21．77pg／ml比较差异非常显著（P＜0．01），与脐血56．35±26．82pg／ml比较，P＜0．05。随呼衰加重NE与E水平均增高，Ⅰ型61．44±33．48与Ⅱ型100．4±22．31（P＜0．05）。NE与PO2呈负相关，r＝－0．414，但无统计学差异。
In this paper, 23 cases of neonatal asphyxia and respiratory failure in children using high performance liquid chromatography - electrochemical detection of norepinephrine (NE) and epinephrine (E), 10 cases of normal umbilical cord blood as the control group, the results showed that asphyxia And respiratory failure hypoxia can lead to increased activity of the sympathetic nervous system, secretion of NE and E significantly increased 87.51 ± 35.31pg / ml acute phase and recovery of 38.55 ± 21.77pg / ml difference was significant (P < 0.01), compared with 56.35 ± 26.82pg / ml cord blood, P <0.05. The levels of NE and E increased with the increase of respiratory failure, type Ⅰ 61.44 ± 33.48 and type Ⅱ 100.4 ± 22.31 (P <0.05). NE and PO2 was negatively correlated, r = -0.414, but no significant difference.