目的　探讨MR快速动态增强在肝癌TACE术后随访中的作用和限度。方法　 2 2例肝癌共 2 4个病灶 ,TACE术后 4～ 9周行MR检查 ,观察病灶在SET1WI和T2 WI上的信号变化及FMPSPGR快速动态增强扫描中的强化表现 ,并行病理对照研究。结果　病灶在T1WI和T2 WI上信号多变。FMPSPGR动态增强早期有强化者 18个 ,无强化者 6个。MR病理对照结果示T1WI上高信号者为肿瘤或出血 ;等信号者为肿瘤、凝固性坏死或炎性细胞浸润 ;低信号者为肿瘤、液化性坏死、凝固性坏死及炎性细胞浸润。T2 WI上高信号者为肿瘤、出血、液化性坏死及炎性细胞浸润 ;等信号者为肿瘤、炎性细胞浸润 ;低信号者为凝固性坏死。动态增强早期有强化者为存活肿瘤 ,无强化区为坏死组织 ;瘤周肿瘤浸润和炎性反应均可表现为延迟强化。结论　FMPSPGR快速动态增强扫描可准确判断TACE术后肿瘤存活和坏死的情况 ,结合SET2 WI可客观评价TACE术后的疗效。 Objective To explore the role and limits of rapid and dynamic enhancement of MR in the follow-up of liver cancer after TACE. Methods A total of 24 lesions were performed in 22 patients with HCC. MR imaging was performed 4 to 9 weeks after TACE. The signal changes on SET1WI and T2WI and the enhanced performance on FMPSPGR rapid dynamic contrast scan were observed. Results The lesions showed variable signals on T1WI and T2WI. In the early stage of dynamic enhancement of FMPSPGR, there were 18 intensifiers and 6 without enhancement. MR pathology control results showed that high signal on T1WI was tumor or hemorrhage; tumors, coagulative necrosis, or inflammatory cell infiltration were observed in the signal; tumors, liquefactive necrosis, coagulative necrosis, and inflammatory cell infiltration were observed in patients with hypointensity. The high signal on T2 WI was tumor, hemorrhage, liquefaction necrosis, and inflammatory cell infiltration; the other signal was tumor and inflammatory cell infiltration; the low signal was coagulative necrosis. In the early stage of dynamic enhancement, those who had fortified were survival tumors, and those without enhanced regions were necrotic tissues. Both tumor infiltration and inflammatory reactions around the tumor could be expressed as delayed enhancement. Conclusions FMPSPGR rapid dynamic contrast-enhanced scanning can accurately determine the survival and necrosis of tumor after TACE, and combined with SET2WI can objectively evaluate the efficacy of TACE.