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目的：从形态学和功能方面探讨外阴营养不良的病因和发病机理。方法：对各型外阴营养不良的酶组织化学与组织学进行对比研究。结果：外阴营养不良的主要病变在真皮层，病变症结在微血管和胶原纤维。真皮内细胞出现程度不一的退行性变、微血管障碍及胶原纤维生成障碍。各型病变的琥珀酸脱氢酶显示较弱，而乳酸脱氢酶相对较强。结论：外阴营养不良病变内氧化水平较低，而醣酵解水平相对较高。该病变可能与细胞代谢的功能紊乱有关。
Objective: To explore the etiology and pathogenesis of vulvar dystrophy from morphological and functional aspects. Methods: The comparative study of histochemistry and histochemistry of various genital malnutrition. Results: The main lesions of vulvar dystrophy in the dermis, lesions in the microvascular and collagen fibers. Dermal cells appear varying degrees of degenerative changes, microvascular disorders and collagen production obstacles. Succinate dehydrogenase showed weak in all types of lesions, while lactate dehydrogenase was relatively strong. Conclusion: Oxidative level in vulvar dystrophy lesion is low, while glycolysis level is relatively high. The lesion may be related to dysfunction of cellular metabolism.