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本文于喀喇昆仑山海拔3700m地区对收治的22例高原昏迷患者在治疗前和治愈后分别检测了血Hb、RBC－SOD、LPO和VitC，并与初入高原的健康青年做对照。结果：高原昏迷患者治愈后RBC－SOD显著低于治疗前（P＜0．01），更低于对照组（P＜0．001）、治愈后LPO较治疗前无显著差异（P＞0．05），但显著高于对照组（P＜0．001）。治愈后VitC显著高于治疗前（P＜0．05），显著低于对照组（P＜0．001）。作者认为，高原昏迷的发生与急性缺氧引发的机体一系列自由基损伤关系十分密切。
Twenty-two patients with plateau coma admitted to the Karakorum Mountains at 3700 m above sea level were tested for blood Hb, RBC-SOD, LPO and VitC before and after treatment, and were compared with healthy youth who entered the plateau. Results: The RBC-SOD in patients with altitude coma was significantly lower than that before treatment (P <0.01) and lower than that in control group (P <0.001). No significant difference was found in LPO after treatment (P> 0.05). 05), but significantly higher than the control group (P <0.001). VitC after treatment was significantly higher than before treatment (P <0.05), significantly lower than the control group (P <0.001). The author believes that the occurrence of plateau coma and acute hypoxia caused by a series of free radical damage is very close to the body.