Lung tissue is very sensitive to ischemia and reperfusion,and the injury mainly manifests as follows: increase in microvascular permeability and pulmonary capillary resistance,with formation of microthrombus and tissue edema,and disordered gas exchange[1]. A number of studies showes that,apoptosis is involved in the pathophysiological mechanism of lung ischemia/reperfusion injury (LIRI)[2-3],and the intervention of apoptosis plays an obvious role in lung injury[4]。