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本实驗观察銻剂靜脉注射及局部灌注后,对刺激压力感受器及化学感受器所引起的反应性的影响,发現: (1) 靜脉注射酒石酸銻鉀每公斤体重20毫克后,能抑制由刺激猫后肢化学感受器所引起的反应性,但对犬的作用不显著。(2) 靜脉注射酒石酸銻鉀每公斤体重20毫克后,能抑制由刺激頸动脉及膀胱压力感受器所引起的反应性,当注射酒石酸銻鉀每公斤体重60毫克时,抑制作用更显著。(3) 用銻剂(1:50,000—25,000)持续灌注人后肢血管可提高刺激后肢化学感受器所引起的反应性,这可以認为是由于微量銻剂長期刺激后肢化学感受器从而提高中樞的兴奋性所致。
The experimental observation of antimony agent intravenous injection and local perfusion on the stimulation of baroreceptors and chemoreceptors caused by the reactivity and found that: (1) intravenous injection of antimony potassium tartrate per kilogram of body weight of 20 mg, can inhibit the stimulation of cats Reactivity induced by hind limb chemoreceptors, but not significant for dogs. (2) Antimony potassium tartrate 20 mg per kg of body weight can inhibit the reactivity caused by stimulation of carotid artery and bladder pressure receptor. When antimony potassium tartrate is injected 60 mg per kilogram of body weight, the inhibitory effect is more remarkable. (3) Continuous perfusion of human hindlimb blood vessels with antimony agents (1: 50,000-25,000) increases the reactivity induced by the stimulation of hind limb chemoreceptors, which is believed to be due to prolonged stimulation of the hindlimb chemoreceptors with trace antimony agents to enhance central excitability Due.