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目的:探讨硫化氢(H2S)预处理保护心肌缺血再灌注损伤作用是否与其抑制过度内质网应激(ERS)引起的心肌细胞凋亡有关。方法:建立大鼠心肌缺血再灌注模型;实验分为:假手术(sham)组、缺血/再灌注(I/R)组和NaHS预处理组。观察分
AIM: To investigate whether the preconditioning of hydrogen sulfide (H2S) protects myocardium against ischemia / reperfusion injury and whether it inhibits cardiomyocyte apoptosis induced by excessive endoplasmic reticulum stress (ERS). Methods: Myocardial ischemia-reperfusion model was established in rats. The experiment was divided into sham group, I / R group and NaHS preconditioning group. Observation points