Abnormal calcium/calmodulin (CaM)-dependent kinase Ⅱ (CaMKⅡ) changes have been shown to be involved in many epileptic models,however,the exact mechanisms
In the present study,the roles of Ca2+/CaM dependent protein kinase Ⅱ (CaMKⅡ) and Calf Intestinal Alkaline Phosphatase (CIP) on the binding of calmodulin